Nature Aging
GLP-1 receptor agonism counteracts omics aging in mice
Pore-forming venoms as a senolytic strategy
Senotoxins target senescence via lipid binding specificity, ion imbalance and lipidome remodeling
CDK3 drives neuron loss in Alzheimer’s disease
Socioeconomic reforms are needed to address disparities for the aging rural-to-urban migrant workforce in China
Evaluating music interventions to treat depression in people living with dementia
Author Correction: Effect of the mitophagy inducer urolithin A on age-related immune decline: a randomized, placebo-controlled trial
Brain neuron-derived WDFY1 induces bone loss
Shear stress governs hematopoietic stem cell fate to promote inflammation-induced aging
Linking single-cell multiomics with GWAS to reveal key regulators of disease risk
Squishing, squeezing and stretching age hematopoietic stem cells
Rejuvenating the aging gut by targeting senescence
Cancer-cell-secreted DDAH1 induces TGF-β1/Smad3 signaling pathway to promote fibrosis and aging in lung
Allosteric activation of a cell-type-specific GPR120 inhibits amyloid pathology of Alzheimer’s disease
Repurposing glucagon-like peptide-1 receptor agonists for the treatment of neurodegenerative disorders
CDK3 induces neuronal death and brain atrophy in Alzheimer’s disease
Lysophosphatidylcholines are associated with amyloidosis in early stages of Alzheimer’s disease
Integrating polygenic signals and single-cell multiomics identifies cell-type-specific regulomes critical for immune- and aging-related diseases
GDF3 promotes adipose tissue macrophage-mediated inflammation via altered chromatin accessibility during aging
Addendum: Aging by the clock and yet without a program
How long will we live? And how much of that time will comprise a healthy life? What is aging, and can we stop or even reverse the aging process? What is the connection between aging and disease? Can we predict the evolving trends in the aging of human populations and prepare our societies for what has been called the Silver Tsunami? These are some of the important questions that the broad field of aging research is trying to address and that together form one of the Grand Challenges of the twenty-first century. The mission of Nature Aging is to provide a unique multidisciplinary, unifying and highly visible publishing platform for the aging-research community. The journal is highly selective yet broad in its coverage, publishing research from across the entire spectrum of the field, ranging from the basic biology of aging to the impact of aging on society. The journal aims to foster interactions among different areas of this diverse field of research and to promote new and exciting ideas within and beyond the research community, to enable synergy and maximize scientific and societal impact.
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