Nature Aging
Lifestyle interventions can save over US $500 million in healthcare spending
The GNPC provides a proteomic resource for biomarker discovery and mechanistic insight in neurodegenerative disease
Disease burden, lifetime healthcare cost and long-term intervention impact projections among older adults in Singapore
Extracellular vesicles from antler blastema progenitor cells reverse bone loss and mitigate aging-related phenotypes in mice and macaques
Single-cell and spatial transcriptomics map senescent vascular cells in arterial remodeling during atherosclerosis in mice
Rethinking inflammaging across human diversity
Characterizing primary and secondary senescence in vivo
Author Correction: mRNA metabolism regulator human antigen R (HuR) regulates age-related hearing loss in aged mice
Insights into bridging disciplines and building measures from the Gateway Exposome Coordinating Center workshop
Author Correction: Large-scale network analysis of the cerebrospinal fluid proteome identifies molecular signatures of frontotemporal lobar degeneration
FOXO3-enhanced mesenchymal progenitor cells impede aging in monkeys
Tau PET positivity marks early clinical transition in Alzheimer’s disease progression
Author Correction: Harnessing a noncanonical vestibular input in the head-direction network to rectify age-related navigational deficits
Association of midlife hearing impairment and hearing aid use with incident dementia: analysis of two UK-based longitudinal cohort studies
DunedinPACNI estimates the longitudinal Pace of Aging from a single brain image to track health and disease
Nonuniversality of inflammaging across human populations
Epigenetic erosion of H4K20me1 induced by inflammation drives aged stem cell ferroptosis
A TFEB–TGFβ axis systemically regulates diapause, stem cell resilience and protects against a senescence-like state
Clusterin drives myeloid bias in aged hematopoietic stem cells by regulating mitochondrial function
Senescent macrophages induce ferroptosis in skeletal muscle and accelerate osteoarthritis-related muscle atrophy
How long will we live? And how much of that time will comprise a healthy life? What is aging, and can we stop or even reverse the aging process? What is the connection between aging and disease? Can we predict the evolving trends in the aging of human populations and prepare our societies for what has been called the Silver Tsunami? These are some of the important questions that the broad field of aging research is trying to address and that together form one of the Grand Challenges of the twenty-first century. The mission of Nature Aging is to provide a unique multidisciplinary, unifying and highly visible publishing platform for the aging-research community. The journal is highly selective yet broad in its coverage, publishing research from across the entire spectrum of the field, ranging from the basic biology of aging to the impact of aging on society. The journal aims to foster interactions among different areas of this diverse field of research and to promote new and exciting ideas within and beyond the research community, to enable synergy and maximize scientific and societal impact.
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