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Janus graphene nanoribbons with localized states on a single zigzag edge
Bidirectional histone monoaminylation dynamics regulate neural rhythmicity
Hippocampal neuronal activity is aligned with action plans
Heritable polygenic editing: the next frontier in genomic medicine?
β-C−H bond functionalization of ketones and esters by cationic Pd complexes
Amino acids and KLHL22 do not activate mTORC1 via DEPDC5 degradation
Author Correction: A deep catalogue of protein-coding variation in 983,578 individuals
Cosmic carnage: planetary rubble spotted at a dying star
A new-year round-up of the science stories you may have missed
Reply to: Amino acids and KLHL22 do not activate mTORC1 via DEPDC5 degradation
Author Correction: Common and rare variant associations with clonal haematopoiesis phenotypes
EBP1 potentiates amyloid β pathology by regulating γ-secretase
Modulating mTOR-dependent astrocyte substate transitions to alleviate neurodegeneration
Quantum mechanics 100 years on: an unfinished revolution
Why kids need to take more risks: science reveals the benefits of wild, free play
Graduate-student stipends in Canada below the poverty line
How quantum mechanics emerged in a few revolutionary months 100 years ago
What will viruses do next? AI is helping scientists predict their evolution
Recollection-related fMRI effects in entorhinal cortex predict longitudinal memory change in healthy older adults
The present study examines whether structural and functional variability in medial temporal lobe (MTL) neocortical regions correlate with individual differences in episodic memory and longitudinal memory change in cognitively healthy older adults. To address this question, older adults were administered a battery of neuropsychological tests on three occasions: the second occasion one month after the first test session, and a third session three years later. Structural and functional MRI data...
Lineage tracing senescence in vivo shows not all senescent cells are created equal
Understanding the impact of senescence on disease is limited by the lack of tools to lineage label senescent cells. In a recent Cell issue, Zhao et al. create mouse models to genetically manipulate and trace p16^(+) cells, identifying contrasting roles for senescent macrophages and endothelial cells (ECs) in liver fibrosis.